Cancer Therapy: Preclinical Carfilzomib-Dependent Selective Inhibition of the Chymotrypsin-like Activity of the Proteasome Leads to Antitumor Activity in Waldenstrom's Macroglobulinemia

نویسندگان

  • Antonio Sacco
  • Monette Aujay
  • Brittany Morgan
  • Abdel Kareem Azab
  • Patricia Maiso
  • Yang Liu
  • Yong Zhang
  • Feda Azab
  • Hai T. Ngo
  • Ghayas C. Issa
  • Phong Quang
  • Aldo M. Roccaro
  • Irene M. Ghobrial
چکیده

Purpose: Primary Waldenstrom’s Macroglobulinemia (WM) cells present with a significantly higher level of the immunoproteasome compared with the constitutive proteasome. It has been demonstrated that selective inhibition of the chymotrypsin-like (CT-L) activity of constitutive-(c20S) and immuno-(i20S) proteasome represents a valid strategy to induce antineoplastic effect in hematologic tumors. We therefore evaluated carfilzomib, a potent selective, irreversible inhibitor of the CT-L activity of the i20S and c20S in

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Carfilzomib-dependent selective inhibition of the chymotrypsin-like activity of the proteasome leads to antitumor activity in Waldenstrom's Macroglobulinemia.

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تاریخ انتشار 2011